NIH R01 · 2025
Project Summary/Abstract Exposure to alcohol during prenatal brain development, perturbs hippocampal development and results in lifelong cognitive deficits in spatial memory. While rodent models have characterized cellular and synaptic changes produced by prenatal alcohol exposure (PAE), the neurophysiological mechanisms in humans remains poorly understood. There is a critical need to identify neural circuitry and brain dynamics involved in spatial memory deficits in humans. Determining the neural mechanisms underlying spatial memory deficits in children with PAE will provide foundational knowledge on how PAE alters brain function and a novel scientific framework allowing for the…
From the public funding record at NIH RePORTER. Describes the funded project, not the reviews below.